Behavioral Neurosciences
Research Laboratory

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Jessica Loweth
 
 

Loweth JA, Li D, Cortright JJ, Wilke G, Jeyifous O, Neve RL, Bayer KU, Vezina P. Persistent reversal of enhanced amphetamine intake by transient CaMKII inhibition. The Journal of Neuroscience 2013; 33: 1411-1416.

Li D, Herrera S, Bubula N, Nikitina E, Palmer AA, Hanck DA, Loweth JA, Vezina P. Casein kinase 1 enables accumbens amphetamine-induced locomotion by regulating AMPA receptor phosphorylation. Journal of Neurochemistry 2011; 118: 237-247.

Loweth JA, Vezina P. Sensitization. Calcium-calmodulin-dependent protein kinase II and the expression of stimulant sensitization. In Olmstead MC, editor, Animal Models of Drug Addiction. Series Neuromethods (W Walz, editor), Volume 53. Totawa, NJ: Humana Press; 2011, p. 191-205.

Singer BF, Loweth JA, Neve RL, Vezina P. Transient viral-mediated overexpression of α-calcium/calmodulin-dependent protein kinase II in the nucleus accumbens shell leads to long-lasting functional upregulation of α-amino-3-hydroxyl-5-methyl-4-isoxazole-propionate receptors: Dopamine type-1 receptor and protein kinase A dependence. European Journal of Neuroscience 2010; 31: 1243-1251.

Loweth JA, Singer BF, Baker LK, Wilke G, Inamine H, Bubula N, Alexander JK, Carlezon WA Jr, Neve RL, Vezina P. Transient overexpression of α-Ca2+/calmodulin-dependent protein kinase II in the nucleus accumbens shell enhances behavioral responding to amphetamine. The Journal of Neuroscience 2010; 30: 939-949.

Loweth JA, Svoboda R, Austin JD, Guillory AM, Vezina P. The PKC inhibitor Ro31-8220 blocks acute amphetamine-induced dopamine overflow in the nucleus accumbens. Neuroscience Letters 2009; 455: 88-92.

Loweth JA, Baker LK, Guptaa T, Guillory AM, Vezina P. Inhibition of CaMKII in the nucleus accumbens shell decreases enhanced amphetamine intake in sensitized rats. Neuroscience Letters 2008; 444: 157-160.

 


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Department of Psychiatry & Behavioral Neurosciences
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Last Update: January 2013